Alkalinity destroysCOVID-19HIVAIDSSARSMALARIAHERPESSYPHILIS

Alkaline supplements destroy Crustaceous shells and Polysaccharide Capsules

The Pycnogonida species, commonly called the spider crab consists of a crustaceous body with an exoskeleton composed of a sugary carbohydrate. In 1917 Oswald Avery of the Rockefeller institute first discovered that some pneumococci were surrounded by a capsule of polysaccharides, a pure carbohydrate sugar shell. In Avery’s first research paper on the subject, he investigated these “specific soluble substances”.

The crustaceous shell of the Pycnogonida is the polysaccharide capsule of the phage virus. In both the large breed Pycnogonida and the microscopic phage virus, the composition and stereochemistry of the lifeform is the same. The exoskeleton of the Pycnogonida is a low-Ph acetic sugar that provides physical protection to the internal gelatinous mass of the species. The exoskeleton becomes tempered at elevated temperatures and is extraordinarily strong. In a parasitic infection of Pycnogonida, the exoskeleton is nearly impenetrable to the immune system of a host.

Although the exoskeleton of the Pycnogonida provides a great physical tensile strength, the chemical composition of low Ph acetic sugar carbohydrates is the species' primary weakness. In an alkaline environment, or when in contact with an alkaline substance, the polysaccharide capsule or exoskeleton of the Pycnogonida completely dissolves.

Upon dissolution of the carbohydrate exoskeleton or polysaccharide capsule of the Pycnogonida species, a gelatinous globule of purple-colored mass of internal organs will remain. This purple gelatinous mass is the Physalia physalis, commonly known as the Portuguese Man O’ War. The Physalia physalis is the large breed of all plasmodium parasites.

Oswald Avery, Rene Dubos, Michal Heidelberger, Fred Griffith, Michael Dawson, Colin Macleod, Maclyn McCarty, and other scientists during and after the great influenza outbreak of 1918 began the study of polysaccharide capsules in pneumococci. Avery coined the term “the sugar-coated microbe”.

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Defeating Phage Viruses with High-Ph Diets

The Pycnogonida can be dissolved in the human body via chemical REDOX reaction with common alkaline substances such as Niacin, Creatine, Zinc, Turmeric, Ginger, Nutmeg, and Colloidal Silver.

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Learn more about REDOX reactions

Alkaline Materials turns the acidic virus into salts

Ancient humans used herbs and supplements to fight off plague, the U.S. military uses them to fight off disease on nuclear powered vessels, and now you can use them at home to fight off COVID-19 and other phage viruses

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Phages can be broken down into purple gelatinous masses

During the great influenza of 1918, it was discovered that the immune system could not attack pneumococci surrounded by capsules, but easily destroyed pneumococci without capsules. Exactly one century before the worldwide epidemic of COVID-19, pneumococci with intact exoskeletons were growing rapidly in the lungs of their hosts and killing within weeks, days, and even hours.

From the Gelatinous Masses come more Phages

In 1928 Fred Griffin published that pneumococcus without capsules were somehow “changing” into pneumococci with capsules. After developing Grave’s disease from working with infectious parasites largely unprotected, Avery developed the current theory on the transformation of pneumococci without capsules into pneumococci with capsules. In 1945 Oswald Avery and Colin McCloud published the paper “Studies on the chemical nature of the substance inducing transformation of pneumococcal types. Induction of transformation by a Desoxyribonucleic Acid Fraction Isolated from Pneumococci Type III” to the “Journal of Experimental Medicine”.

Avery died so we could kill Phage Viruses

In 1928 Oswald Avery developed Grave’s disease during years of unprotected research into pneumococci. Avery at over 40 Years of age worked during the worst virus outbreak in modern world history. Avery worked in simplified labs with a single porcelain sink, worktables, Bunsen burners, racks of test tubes, mason jars, Petri dishes, droppers, pipettes, platinum loops, incubators, vacuum pumps, and centrifuges. During the great influenza outbreak, Avery performed all work on the same tabletop, incubating virus samples, inoculating animals, and bleeding or dissecting infected animals. Avery walked to the laboratory every day wearing the same grey jacket and changed into a light tan or white lab coat as his only protection. Avery’s assistant Rene Dubos observed that “He made little effort to follow modern trends in science … In the lab he was limited to a rather narrow range of techniques, which he rarely changed and to which he added little.” 

-Oswald Theodore Avery, 1877-1955 “Biological Memoirs of Fellows of the Royal Society (1956), 40.

The current theory of the formation of Pneumococcus with capsules from those without capsules is accepted as “The inducing substance has been likened to a gene, and the capsular antigen which is produced in response to it has been regarded as a gene product”. The current theory accepted by modern science is that genes of pneumococcus are written to repair or grow new capsules, and that DNA just repairs the virus. Observations of large breed Pycnogonida provide modern scientists a better perspective on theories formed from over microscopes a century ago. Capsules cannot regrow on unencapsulated viruses (plasmodium parasites) to become re-encapsulated (Phage Viruses), but plasmodium parasite and phage viruses can breed many encapsulated viruses.

In 1928 Fred Griffith published that when pneumococci without capsules were incubated with seemingly dead pneumococci with capsules intact, he believed that the pneumococci without capsules somehow transformed into pneumococci with capsules intact. Oswald Avery dedicated his life trying to figure out how specimens of pneumococci were transformed.

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REDOX Reactions turn Phage Viruses into Plasmodium Parasites

When an alkaline environment or substance reduces the oxidized polysaccharide hydrocarbonate capsulized exoskeleton of the Pycnogonida, the internal mass which remains is the plasmodium parasite or Physalia physalis, commonly referred to as the Portuguese Man O’ War.

In a liquid or aquatic environment, the coil structures which are externally visible in the Physalia physalis species immediately become intertwined in a randomly contorted mess. The randomly intertwined bundle of coils hangs from a balloon-like body which expands after losing the pressurized capsule or exoskeleton. These intertwined coil structures can be seen contracting and expanding beneath the surface of the water from the Physalia physalis.

Sections of the Pycnogonida's legs, body, head, tail, and ovigars dissolve via REDOX reactions quickly within minutes when in contact with a reducing agent. Complex coil groups once anchored to points of the exoskeleton become one big hanging mess. The reduction of the Pycnogonida into the Physalia physalis can be performed easily in a level 4 laboratory and has been observed on Navy ships, in nuclear reactors, and in the natural environment of the Earth’s oceans. What has never been performed or observed in any laboratory, industrial, or the natural environment is the unraveling of tangled coils by the Physalia physalis and regrowth of an overly complex exoskeleton with anchoring points to each individual coil needed to support functions of movement, hunting, feeding, and life. 

Dissolving the oxidized shell, exoskeleton, or polysaccharide capsule of the large breed Pycnogonida or microscopic phage virus, leaving the large breed Physalia physalis or microscopic Plasmodium parasite is much like cracking the shell of an egg, removing the shell from the egg, and scrambling its contents and it just cannot be undone. The damage done to the coils and anchor points within the exoskeleton and the bloating of the bladder of the resulting Physalia physalis during a REDOX reaction is simply too violent to the delicate and complex structure of the species to ever grow back under and condition.

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Phages are stored within Plasmodium Parasites

Unknowing that the viruses they were studying were sea creatures, scientists such as Oswald Avery and Fred Griffith published the current theory that pneumococci species whose capsule has been dissolved could somehow be observed under the microscope transforming into species with intact capsules.

Modern Observations of Physalia Physalis without an oxidized exoskeleton or hydrocarbonate polysaccharide capsule have revealed that the birth of new spawns of Pycnogonida from Physalia Physalis tentacles is the only method available to produce specimen with an intact capsule from populations of the specimen without capsules. Physalia physalis give birth to new Pycnogonida with intact capsules in the form of nematocysts which spawn from select coils hanging from the blue-purple gelatinous mass.

In a natural environment or laboratory tank, nematocysts hatch from gonopores within the exposed coils and fall to the seafloor or tank floor. Alternatively, coils that come in contact with a species of prey can inject nematocysts into a host via a “sting” which allows the infant nematocysts to grow as a parasite infection, further spreading the species and continuing its lifespan.

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Plasmodium Parasites can spread in a mildly alkaline environment

A Pycnogonida which loses its protective structure may release millions to billions of smaller Physalia physalis and Plasmodium parasites which all simultaneously lose their protective structures from contact with a common reducing agent. Each generation of damaged Physalia physalis has within its reproductive coils, new generations of intact Pycnogonida. Larval Pycnogonida within Physalia physalis reproductive coils are called Nematocysts. Each generation of Pycnogonida produces new spawns from its gonopores, simultaneously.

This biological method of exponential reproduction is how viruses spread so far and so fast in short periods. A large breed of Pycnogonida enters a host-prey and immediately releases all generations of spawn into the host’s body to spread, grow, and reproduce. This method of parasitic infection is highly effective at surviving on a host’s blood, and organs until the species can be spread throughout the host species’ population.

Plasmodium parasites give birth to phage viruses with intact capsules

Between 1921 and 1945 scientists believed they were seeing a transformation of pneumococci without capsules into pneumococci with capsules during lab experiments. Modern observations made in laboratories, industrial, military, and natural environments show what in fact, pneumococci without capsules seen by scientists like Oswald Avery and Fred Griffith were spawning new Pycnogonida as phage viruses before being decayed and broken down by death.

A transformation of viral cells was not a reality. A cycle of birth to new generations of virus cells before death and decay following the natural cycle is the truth and a quite common reality among living populations of species.

With an intact exoskeleton (capsule), nematocysts exit pycnogonida gonopores as phages

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Exponential Breeding

In an intact Pycnogonida, specialized coils lead to gonopores in the legs of the species. Gonopores are exposed openings that give birth to exponentially polyping generations of new Pycnogonida. A single gonopore exists on each section of every leg, between each joint. With eight legs, each having five sections, the Pycnogonida has 40 gonopores. Every gonopore can simultaneously spawn a single Pycnogonida. Every generation of newly spawned Pycnogonida grows from its parent's gonopores until it is released, often occurring only in the presence of survivable habitat or targeted host for parasite invasion. Each generation of spawned Pycnogonida is capable of spawning newer generations from its gonopores, even while still attached and growing from its own parent’s leg.

In effect, a first-generation Pycnogonida with a 3-meter leg span may spawn 40 attached Pycnogonida offspring with leg spans of 2 decimeters. 40 second-generation 2dm Pycnogonida may simultaneously spawn 1600 attached Pycnogonida with a 2cm leg span. The 1600 third-generation Pycnogonida may spawn 64,000 attached Pycnogonida with a 2mm leg span. The 64,000 fourth-generation Pycnogonida may spawn 2,560,000 attached Pycnogonida with a 200-micrometer leg span. The 2,560,000 fifth-generation Pycnogonida may spawn 102,400,000 attached Pycnogonida with a leg span of 20 micrometers. The 102,400,000 sixth-generation Pycnogonida may spawn 4,096,000,000 Phage virus sized Pycnogonida.