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Viral Infection effects on the Nervous System
- Category: Phage Viruses
The Process of Demyelination and Acquired Diseases Pycnogonida and their associated viral infections attack, destroy, and manipulate the brain, spine, and nervous system of hosts that they infect. During autopsies of hosts of lethal viruses, the proteins, cytokines, macrophages, granulocytes, plasmodium parasites, and their various Genuses are found within the white matter of the cerebrum, in the meninges which surrounds the brain, and within the spinal cord. The fatty lipid layers which cover and insulate portions of the brain are called Myelin. Myelin to the function of the nervous system is like the silicone insulation which surrounds and insulates electrical copper wires and cables or used in circuit boards to separate individual electric components.
Myelin protects and isolates electrical signals produced within the brain which travel down the spinal cord and throughout the nervous system. To viruses, Mylin has only one function, being a source of available calories as a food supply. In the brain, virus infections target and consume the fatty lipid content which surrounds and insulates neurons, dendrites, and nerve bundles contained within the white matter of the cerebrum. When Myelin which protects the brain is consumed in a process called Demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are common systems of Demyelination resemble symptoms of decay found on insulation within electrical systems and devices from rodent infestations.
Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech found as epidemic diseases such as M.S., Parkinson’s, and epilepsy. Demyelination can only be halted and reversed via removal of the virus infection from the body and consumption of massive volumes of regenerative supplements such as collagen, MSM, and glucosamine, and amino acids.
Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. While modern science and medicine offer multiple prescriptions and medications to treat Demyelination in hosts of disease, only high-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.
Pycnogonida are a highly intelligent, hive-minded species that uses electrical signals generated from Tritium beta decay as a method of communication. Coiled nervous systems found within the species use double latch gates to keep quantum bonds between generations of the species. Physical breakage between a child Pycnogonida’s nervous system and the gonopore of its parent leaves behind chemical bonds which can transmit electrical signals throughout offspring exponentially produced by generations of the species. Electrical signals, data, information, communication, and knowledge received by a signal Pycnogonida is available to a neural network which is expanded throughout every connected generation of the species.
The neural network available to Pycnogonida is larger than any other species found on earth, except possibly for another yet Unkown species of polyping medusa found within the ocean. Exponentially reproducing Pycnogonida parasites that have gained access to an infected host’s brain and neural network quickly learn (or already have knowledge) of the language of signal code used by the human nervous system to command the executive functions. In “Acquired” diseases, a host or host has been demyelinated and the viral infection has acquired the ability to intercept, interpret, and send electrical signals to and from an infected brain of an infected host.
A host of an acquired disease becomes a puppet of varying degrees of control to a type of “middle-man attack" by the virus population. Acquired hosts stand for the well-documented appearance of schizophrenia, delirium, mental inertia, physical prostration, psychosis, mental collapse, mental disturbances, and general insanity documented after the appearance of a majority of historical influenza and virus outbreaks. At the height of the largest and deadliest virus epidemic in modern recorded history, the influenza outbreak of 1918 brought in reports of outbreak induced mental disturbances from all over the world:
At the U.S. Army’s Walter Reed Hospital, physicians Dr. Egbert Fell reported via the June 1919 issue of the Journal of the American Medicine Association, that patients possessed “Delirium occurring at the height of the Disease”, and that these symptoms did not “clear with cessation of fever”. Patients developed severe mental health symptoms during influenza infections which did not pass even when other symptoms of the virus had. Doctors in Britain reported “profound mental inertia with intense physical prostration. Delirium has been very common … It has varied from more confusion of ideas through all grades on intensity up to maniacal excitement”. Influenza patients were having and communicating wild ideas that may not have existed prior to a virus infection. These hosts became motivated by these ideas from an unknown origin to the point that they became excited and overcame physical inhibitions in response. These symptoms resulted from the presence of alien virus infections.
The Processes of Viral Uncoating
- Category: Phage Viruses
Process of rapid consumption of calories at an exponential growth rate Once an invading population of Phage viruses has successfully invaded body cavities or the epidermal layer of their targeted prey, the parasite begins consuming their host. The processes of decay which results from parasitic infection by Pycnogonida become the symptoms from which modern medicine creates cases for various diseases. A host becomes not only a source of calories for the parasite but also an incubator for their new spawn.
Pycnogonida dissolve the body from within, while Tritium shed from the species becomes a multiplier for the rate of decay within the host. At a microscopic level, the process of a parasite virus dissolving a host cell is called “uncoating”. Uncoating begins with a loss of the host’s calories at an exponentially increasing rate. The Pycnogonida is a tophaphagous predator which targets healthy areas within the host’s body from which it can leach available calories from the host. A first-generation invading parasite will consume a host’s calories in excess, grow, and produce as many strong offspring as possible. Second-generation invading parasites produced from the first generations will do the same thing.
The Process of Forming a Vesicle
- Category: Phage Viruses
The Process of Forming a Vesicle As soon as Phage viruses have engulfed a cell to complete the process of “absorption”, powerful claws and jaws of the phage viruses begin tearing into the cell and looking for a way inside. Phages tear open cell membranes and/or find existing cavities that attack aggressively. Once an entry point is created or found, more phages begin attacking the opening and digging themselves into pockets called “vesicles”. The vesicle is used as a tophaphagous feeding point for the parasitic phage viruses to drain calories from their new host. Once vesicles are formed in a host cell, the cavity or pocket expands as the host cell is consumed by the invading phage viruses and plasmodium parasites.
Pycnogonida invade and enter human bodies or other species using methods identical to tactics of cell invasion used by their infant phage viruses at the microscopic level. Pycnogonida target body cavities such as the mouth, nose, ears, eyes, anus, and genitals. Pycnogonida enter the mouth or nose, then use the lungs, throat, sinuses, and the stomach as the parasite’s vesicles to draw blood from within their new host. Areas of a host’s epidermal layer that is ripped open by the claws and jaws of the Pycnogonida or stung by nematocysts from gonopores on the underside of a Pycnogonida's legs or physalia physalis' tentacles quickly become vesicles for parasite infection.
Lesions formed by species of Pycnogonida are recognizable symptoms of certain smallpox, leprosy, herpes, HIV, Equestrian Encephalitis, Malaria, Coronavirus, and multiple influenza infections. Physalia Physalis is commonly known as the “Portuguese Man O’ War” and their microscopic scale infant plasmodium parasites create vesicles via epidermal injection of nematocysts into a targeted host. Physalia Physalis and Plasmodium parasites alike wrap their prey up with their exposed tentacles. Once these parasites have a grip on a target body or cell, coils that once led to gonopores in the exoskeleton of their previous form (Pycnogonida) are used to inject nematocysts (infant Pycnogonida) directly into an epidermal layer or cell membrane.
Infant Pycnogonida, referred to as “Nematocysts” use a direct coil to epidermal or membrane contact to burrow into a new host much like a blood-sucking tick. Portuguese Man O’ War are deadly to humans and other species if the genus of Pycnogonida which dissolved to form the "jellyfish in appearance" parasite is a type-A or another extremely aggressive variety. The following videos feature a tribal method for removal of Pycnogonida which have created vesicles in the throats of a man and woman in Cuba. This method of removal does not remove the viral load which is left by the Pycngonida within the throat and leaves a dangerous amount of the Pycnogonida’s remains left in the body. These people do not fully understand what the Pycnogonida parasite is, or how they were infected. They know that they have some type of “insect” inside them and want to get it out. Pycnogonida Forming A Vesicle In A Female Vagina
The Process of Virus Absorption
- Category: Phage Viruses
The Process of Virus Absorption The current theory on virus “absorption" onto healthy cells involves the use of “spike-shaped” protuberances on the viruses called “hemagglutinin” to bind and latch onto sialic acid “receptors" on the targeted cell-like grappling hooks. As the theory continues: as more hemagglutinin binds to more sialic receptors on the cell, the virus adheres to the body of the targeted cell. The reality of absorption is: like any other predator in the wild, the Pycnogonida must grip onto areas on its prey where its claws will fit. Once a Pycnogonida gets a hold of a host with several of its eight legs, the predator will quickly grab a hold of the host with the rest of its appendages and make aggressive maneuvers to breach its prey.
On both living bodies and living cells, tactics of predatorial approach by the Pycnogonida species are the same. During microscopic infancy, phage viruses 1/10,000th of a millimeter in diameter are doing the same thing to cells that a 3dm diameter Pycnogonida will do to a fully grown person. Microscopy is never perfect, as scientists are forced to publish explanations for objects, lifeforms, actions, activities, and processes that they cannot see in clear focus. Fancy terminology like hemagglutinin, sialic acid receptors, and binding of the two only supply confusion to the fact that extremely aggressive eight-legged spider-like creatures are jumping onto cells and wrapping their legs around their target to grab hold wherever their claws will fit. Pycnogonida in the wild or running loose in human environments are witnessed using eight legs to crawl, run, leap, swim, and climb toward capturing a host suitable for parasitic infection and reproduction.
Suitable hosts at any scale include prey with an acidic or low-Ph level, which will not break down and cook off the cell via acid-base REDOX reaction. Hive-minded Pycnogonida are highly intelligent and will use all available senses to look for an available host which will not reduce the virus.
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